2006/12 + 2022/23: Murray Bridge (South Australia) – Trihalomethanes, Bromodichloromethane, Total Haloacetic Acids, Chloroketones, Chloral Hydrate

Murray Bridge (South Australia) – Trihalomethanes

Breaches to Australian Drinking Water Guidelines Levels Only

9/03/2011 Murray Bridge Swanport Rd Trihalomethanes – Total 296 ug/L

10/03/2011 Murray Bridge Mitchell Av Trihalomethanes – Total 285 ug/L

Trihalomethanes Australian Guideline Level 250μg/L (0.25mg/L)

Why and how are THMs formed?
“When chlorine is added to water with organic material, such as algae, river weeds, and decaying leaves, THMs are formed. Residual chlorine molecules react with this harmless organic material to form a group of chlorinated chemical compounds, THMs. They are tasteless and odourless, but harmful and potentially toxic. The quantity of by-products formed is determined by several factors, such as the amount and type of organic material present in water, temperature, pH, chlorine dosage, contact time available for chlorine, and bromide concentration in the water. The organic matter in water mainly consists of a) humic substance, which is the organic portion of soil that remains after prolonged microbial decomposition formed by the decay of leaves, wood, and other vegetable matter; and b) fulvic acid, which is a water soluble substance of low molecular weight that is derived from humus”. Source: https://water.epa.gov/drink/contaminants/in

Murray Bridge (South Australia) – Bromodichloromethane

2022/23: Murray Bridge (South Australia) Bromodichloromethane 76ug/L (max), 47.53ug/L (av. 2022/23)

WHO Guideline level BDCM: 60ug/L (Australian Guideline for BDCM is included in the Trihalomethane (THM) combined total of BDCM, Chloroform, Dibromochloromethane and Bromoform. THM guideline is 250ug/L)

“Carcinogenicity : Bromodichloromethane is reasonably anticipated to be a human carcinogen based on sufficient evidence of carcinogenicity from studies in experimental animals.
Cancer Studies in Experimental Animals: Oral exposure to bromodichloromethane caused tumors at several different tissue sites in mice and rats. Administration of bromodichloromethane by stomach tube caused benign and malignant kidney tumors (tubular-cell adenoma and adenocarcinoma) in male mice and in rats of both sexes, benign and
malignant liver tumors (hepatocellular adenoma and carcinoma) in female mice, and benign and malignant colon tumors (adenomatous polyps and adenocarcinoma) in rats of both sexes (NTP 1987, ATSDR 1989, IARC 1991, 1999).

Murray Bridge (South Australia) – Total Haloacetic Acids

8/2/22: Murray Bridge (South Australia) Total Haloacetic Acid (HAA 9) 123ug/L (max) 84ug/L (av. 2021/22)

7/2/23: Murray Bridge (South Australia) Total Haloacetic Acid (HAA 9) 165ug/L (max) 124ug/L (av. 2022/23)

“Chloroacetic acids are produced in drinking water as by-products of the reaction between chlorine and naturally occurring humic and fulvic acids. Concentrations reported overseas range up to 0.16mg/L and are typically about half the chloroform concentration. The chloroacetic acids are used commercially as reagents or intermediates in the preparation of a wide variety of chemicals. Monochloroacetic acid can be used as a pre-emergent herbicide, dichloroacetic acid as an ingredient in some pharmaceutical products, and trichloroacetic acid as a herbicide, soil sterilant and antiseptic.” Australian Drinking Water Guidelines – National Health and Medical Research Council

Murray Bridge (South Australia) – Chloroketones >10ug/L

7/2/23: Murray Bridge (South Australia): 1,1,1-trichloropropan-2-one 10.1ug/L

Data are inadequate to set guideline values for chloroketones in drinking water.
The chloroketones are produced in drinking water as by-products of the reaction between naturally occurring organic matter and chlorine. No data are available on other sources or uses for these compounds. Concentrations of chloroketones in drinking water reported overseas are very low and are estimated at less than 0.01 mg/L.

In major Australian reticulated supplies 1,1,1-trichloropropanone has been recorded in concentrations up to 0.02 mg/L, but it is usually below the limit of determination of 0.0005 mg/L. No data are available for other chloroketones.

The presence of chloroketones in drinking water can be minimised by removing naturally occurring organic matter from the source water, by reducing the amount of chlorine added, or by the use of alternative disinfectants.” 2011 ADWG

Murray Bridge (South Australia) – Chloral Hydrate

4/1/06 Murray Bridge  Chloral Hydrate 25ug/L

5/4/06 Murray Bridge  Chloral Hydrate 28.6ug/L

18/11/10 Murray Bridge  Chloral Hydrate 22.2ug/L

10/3/11 Murray Bridge  Chloral Hydrate 27.8ug/L

7/3/12 Murray Bridge  Chloral Hydrate 24.6ug/L

Chloral hydrate is a disinfection by-product, arising from chlorination of water containing naturally occurring organic material (NOM). Chloral hydrate is a sedative and hypnotic drug. Long-term use of chloral hydrate is associated with a rapid development of tolerance to its effects and possible addiction as well as adverse effects including rashes, gastric
discomfort and severe renal, cardiac and hepatic failure.

2004 Australian Drinking Water Guideline: Trichloroacetaldehyde (chloral hydrate): 0.02mg/L

2011 Australian Drinking Water Guideline: Trichloroacetaldehyde (chloral hydrate): 0.1mg/L